Blockade of Nuclear Factor of Activated T Cells Activation Signaling Suppresses Balloon Injury-induced Neointima Formation in a Rat Carotid Artery Model
Open Access
- 1 April 2005
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 280 (15), 14700-14708
- https://doi.org/10.1074/jbc.m500322200
Abstract
No abstract availableKeywords
This publication has 48 references indexed in Scilit:
- Forthcoming issueSeminars in Oncology, 2004
- A Novel Role for Nuclear Factor of Activated T Cells in Receptor Tyrosine Kinase and G Protein-coupled Receptor Agonist-induced Vascular Smooth Muscle Cell MotilityPublished by Elsevier ,2004
- Celecoxib, a Cyclooxygenase-2 Inhibitor, Reduces Neointimal Hyperplasia Through Inhibition of Akt SignalingCirculation, 2004
- NFATc3 and NFATc4 Are Required for Cardiac Development and Mitochondrial FunctionCirculation Research, 2003
- A Constitutively Active NFATc1 Mutant Induces a Transformed Phenotype in 3T3-L1 FibroblastsJournal of Biological Chemistry, 2003
- Combinatorial Control of Smooth Muscle–Specific Gene ExpressionArteriosclerosis, Thrombosis, and Vascular Biology, 2003
- A potential role for nuclear factor of activated T-cells in receptor tyrosine kinase and G-protein-coupled receptor agonist-induced cell proliferationBiochemical Journal, 2002
- TNF-α–Induced Migration of Vascular Smooth Muscle Cells Is MAPK DependentHypertension, 1999
- TRANSCRIPTION FACTORS OF THE NFAT FAMILY:Regulation and FunctionAnnual Review of Immunology, 1997
- Platelet-derived growth factor promotes smooth muscle migration and intimal thickening in a rat model of balloon angioplasty.Journal of Clinical Investigation, 1992