The JAK2V617F mutation is acquired secondary to the predisposing alteration in familial polycythaemia vera
Open Access
- 10 August 2005
- journal article
- Published by Wiley in British Journal of Haematology
- Vol. 130 (5), 800-801
- https://doi.org/10.1111/j.1365-2141.2005.05683.x
Abstract
No abstract availableKeywords
This publication has 6 references indexed in Scilit:
- A Gain-of-Function Mutation ofJAK2in Myeloproliferative DisordersNew England Journal of Medicine, 2005
- Activating mutation in the tyrosine kinase JAK2 in polycythemia vera, essential thrombocythemia, and myeloid metaplasia with myelofibrosisCancer Cell, 2005
- A unique clonal JAK2 mutation leading to constitutive signalling causes polycythaemia veraNature, 2005
- Acquired mutation of the tyrosine kinase JAK2 in human myeloproliferative disordersThe Lancet, 2005
- Clonal hematopoiesis in familial polycythemia vera suggests the involvement of multiple mutational events in the early pathogenesis of the diseaseBlood, 2003
- Familial polycythemia vera with Budd–Chiari syndrome in childhoodBritish Journal of Haematology, 2003