Constitutive phosphoinositide 3-kinase activation in acute myeloid leukemia is not due to p110δ mutations
- 8 December 2005
- journal article
- Published by Springer Nature in Leukemia
- Vol. 20 (2), 374-376
- https://doi.org/10.1038/sj.leu.2404054
Abstract
No abstract availableKeywords
This publication has 9 references indexed in Scilit:
- Mutant PIK3CA promotes cell growth and invasion of human cancer cellsCancer Cell, 2005
- Essential role for the p110 isoform in phosphoinositide 3-kinase activation and cell proliferation in acute myeloid leukemiaBlood, 2005
- Phosphatidylinositol 3-kinase mutations identified in human cancer are oncogenicProceedings of the National Academy of Sciences, 2005
- High Frequency of Mutations of the PIK3CA Gene in Human CancersScience, 2004
- Biologic and clinical significance of the FLT3 transcript level in acute myeloid leukemiaBlood, 2004
- FLT3 ligand causes autocrine signaling in acute myeloid leukemia cellsBlood, 2004
- Survival of acute myeloid leukemia cells requires PI3 kinase activationBlood, 2003
- Phosphoinositide 3-kinases: A conserved family of signal transducersTrends in Biochemical Sciences, 1997
- p110δ, a novel phosphoinositide 3-kinase in leukocytesProceedings of the National Academy of Sciences, 1997