Endoglin Promotes Transforming Growth Factor β-mediated Smad 1/5/8 Signaling and Inhibits Endothelial Cell Migration through Its Association with GIPC
Open Access
- 1 November 2008
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 283 (47), 32527-32533
- https://doi.org/10.1074/jbc.m803059200
Abstract
No abstract availableKeywords
This publication has 35 references indexed in Scilit:
- ALK5- and TGFBR2-independent role of ALK1 in the pathogenesis of hereditary hemorrhagic telangiectasia type 2Blood, 2008
- Activin receptor‐like kinase 1 inhibits human microvascular endothelial cell migration: Potential roles for JNK and ERKJournal of Cellular Physiology, 2007
- Identification of BMP9 and BMP10 as functional activators of the orphan activin receptor-like kinase 1 (ALK1) in endothelial cellsBlood, 2006
- Interaction and functional interplay between endoglin and ALK‐1, two components of the endothelial transforming growth factor‐β receptor complexJournal of Cellular Physiology, 2005
- Endoglin Regulates Cytoskeletal Organization through Binding to ZRP-1, a Member of the Lim Family of ProteinsJournal of Biological Chemistry, 2004
- A Novel Mechanism for Regulating Transforming Growth Factor β (TGF-β) SignalingJournal of Biological Chemistry, 2001
- Role of Transforming Growth Factor β in Human DiseaseNew England Journal of Medicine, 2000
- Role of Endoglin in Cellular Responses to Transforming Growth Factor-βPublished by Elsevier ,1998
- TGF-beta receptor-mediated signalling through Smad2, Smad3 and Smad4The EMBO Journal, 1997
- Mechanism of activation of the TGF-β receptorNature, 1994