Twenty Years of the Alzheimer’s Disease Amyloid Hypothesis: A Genetic Perspective
Top Cited Papers
- 1 February 2005
- Vol. 120 (4), 545-555
- https://doi.org/10.1016/j.cell.2005.02.008
Abstract
No abstract availableKeywords
This publication has 101 references indexed in Scilit:
- Genetic variants in a haplotype block spanningIDE are significantly associated with plasma A?42 levels and risk for Alzheimer diseaseHuman Mutation, 2004
- Nicastrin gene in familial and sporadic Alzheimer's diseaseNeuroscience Letters, 2003
- Genetic variation in a haplotype block spanningIDE influences Alzheimer diseaseHuman Mutation, 2003
- Genetic association of Alzheimer's disease with multiple polymorphisms in alpha-2-macroglobulinHuman Molecular Genetics, 2003
- Reconstitution of γ-secretase activityNature Cell Biology, 2003
- Nectin-1α, an Immunoglobulin-like Receptor Involved in the Formation of Synapses, Is a Substrate for Presenilin/γ-Secretase-like CleavageJournal of Biological Chemistry, 2002
- The Amyloid Precursor Protein Locus and Very-Late-Onset Alzheimer DiseaseAmerican Journal of Human Genetics, 2001
- BACE Maps to Chromosome 11 and a BACE Homolog, BACE2 , Reside in the Obligate Down Syndrome Region of Chromosome 21Science, 1999
- Increased amyloid beta-peptide deposition in cerebral cortex as a consequence of apolipoprotein E genotype in late-onset Alzheimer disease.Proceedings of the National Academy of Sciences, 1993
- Segregation of a missense mutation in the amyloid precursor protein gene with familial Alzheimer's diseaseNature, 1991