Persistent Increase in Chromosome Instability in Lung Cancer: Possible Indirect Involvement of p53 Inactivation
- 31 October 2001
- journal article
- Published by Elsevier in The American Journal of Pathology
- Vol. 159 (4), 1345-1352
- https://doi.org/10.1016/s0002-9440(10)62521-7
Abstract
No abstract availableKeywords
This publication has 27 references indexed in Scilit:
- Molecular analysis of the mitotic checkpoint genes BUB1, BUBR1 and BUB3 in human lung cancersCancer Letters, 2001
- Search for in vivo somatic mutations in the mitotic checkpoint gene, hMAD1, in human lung cancersOncogene, 1999
- The mitotic machinery as a source of genetic instability in cancerSeminars in Cancer Biology, 1999
- Identification of frequent impairment of the mitotic checkpoint and molecular analysis of the mitotic checkpoint genes, hsMAD2 and p55CDC, in human lung cancersOncogene, 1999
- Prevention of mammalian DNA reduplication, following the release from the mitotic spindle checkpoint, requires p53 protein, but not p53-mediated transcriptional activityOncogene, 1998
- Mitotic Arrest: Mad2 Prevents Sleepy from Waking Up the APCScience, 1998
- The spindle assembly checkpointCurrent Opinion in Cell Biology, 1996
- Abnormal Centrosome Amplification in the Absence of p53Science, 1996
- Pericentrin, a highly conserved centrosome protein involved in microtubule organizationCell, 1994
- Cell biology and molecular biology of small cell and non-small cell lung cancerCurrent Opinion in Oncology, 1990