NKG2D-Deficient Mice Are Defective in Tumor Surveillance in Models of Spontaneous Malignancy
Top Cited Papers
- 1 April 2008
- Vol. 28 (4), 571-580
- https://doi.org/10.1016/j.immuni.2008.02.016
Abstract
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This publication has 64 references indexed in Scilit:
- Magnetic resonance imaging and histopathological characterization of prostate tumors in TRAMP mice as model for pre‐clinical trialsThe Prostate, 2006
- Expression and function of NKG2D in CD4+ T cells specific for human cytomegalovirusEuropean Journal of Immunology, 2006
- Therapy-induced antibodies to MHC class I chain-related protein A antagonize immune suppression and stimulate antitumor cytotoxicityProceedings of the National Academy of Sciences, 2006
- Generation of Antitumor Responses by Genetic Modification of Primary Human T Cells with a Chimeric NKG2D ReceptorCancer Research, 2006
- Activation of the DNA damage checkpoint and genomic instability in human precancerous lesionsNature, 2005
- DNA damage response as a candidate anti-cancer barrier in early human tumorigenesisNature, 2005
- Cancer immunoediting: from immunosurveillance to tumor escapeNature Immunology, 2002
- Deletion of the Igκ Light Chain Intronic Enhancer/Matrix Attachment Region Impairs but Does Not Abolish VκJκ RearrangementImmunity, 1996
- The c-myc oncogene perturbs B lymphocyte development in Eμ-myc transgenic miceCell, 1986
- The c-myc oncogene driven by immunoglobulin enhancers induces lymphoid malignancy in transgenic miceNature, 1985