Dimethylfumarate Induces Immunosuppression via Glutathione Depletion and Subsequent Induction of Heme Oxygenase 1
- 1 April 2007
- journal article
- Published by Elsevier in Journal of Investigative Dermatology
- Vol. 127 (4), 835-845
- https://doi.org/10.1038/sj.jid.5700686
Abstract
No abstract availableKeywords
This publication has 79 references indexed in Scilit:
- Heme oxygenase-1 expression inhibits dendritic cell maturation and proinflammatory function but conserves IL-10 expressionBlood, 2005
- Fumaric acid esters in severe psoriasis, including experience of use in combination with other systemic modalitiesBritish Journal of Dermatology, 2004
- Glutathione in Defense and SignalingAnnals of the New York Academy of Sciences, 2002
- Novel immunotherapies for psoriasisTrends in Immunology, 2002
- Heme Oxygenase: Recent Advances in Understanding Its Regulation and RoleProceedings of the Association of American Physicians, 1999
- Influence of monomethylfumarate on monocytic cytokine formation--explanation for adverse and therapeutic effects in psoriasis?Archives of Dermatological Research, 1997
- Selective stimulation of T helper 2 cytokine responses by the anti‐psoriasis agent monomethylfumarateEuropean Journal of Immunology, 1996
- Antipsoriatic effect of fumaric acid derivativesJournal of the American Academy of Dermatology, 1994
- Normal and Heat‐Induced Patterns of Expression of Heme Oxygenase‐1 (HSP32) in Rat Brain: Hyperthermia Causes Rapid Induction of mRNA and ProteinJournal of Neurochemistry, 1992
- Glutathione monoethyl ester: Preparation, uptake by tissues, and conversion to glutathioneArchives of Biochemistry and Biophysics, 1985