Hypoxia Enhances the Expression of Follistatin-like 3 in Term Human Trophoblasts
- 31 January 2008
- Vol. 29 (1), 51-57
- https://doi.org/10.1016/j.placenta.2007.09.001
Abstract
No abstract availableKeywords
This publication has 49 references indexed in Scilit:
- FSTL3 deletion reveals roles for TGF-β family ligands in glucose and fat homeostasis in adultsProceedings of the National Academy of Sciences, 2007
- IGFBP1 and Follistatin-like 3 Genes are Significantly Up-regulated in Expression Profiles of the IUGR PlacentaPlacenta, 2006
- Antagonists of activin signaling: mechanisms and potential biological applicationsTrends in Endocrinology & Metabolism, 2005
- Human endometrium and decidua express follistatin-related gene (FLRG) mRNA and peptideMolecular and Cellular Endocrinology, 2004
- Cellular adaptation to hypoxia: O2‐sensing protein hydroxylases, hypoxia‐inducible transcription factors, and O2‐regulated gene expressionThe FASEB Journal, 2002
- Transcription activation of FLRG and follistatin by activin A, through Smad proteins, participates in a negative feedback loop to modulate activin A functionOncogene, 2002
- Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT MethodMethods, 2001
- FLRG, an activin-binding protein, is a new target of TGFβ transcription activation through Smad proteinsOncogene, 2001
- Follistatin: A Multifunctional Regulatory ProteinFrontiers in Neuroendocrinology, 1998
- Functional analysis of activins during mammalian developmentNature, 1995