Contact Activation in the Extrinsic Blood Clotting System
- 1 January 1967
- journal article
- research article
- Published by Georg Thieme Verlag KG in Thrombosis and Haemostasis
- Vol. 18 (03/04), 525-531
- https://doi.org/10.1055/s-0038-1655061
Abstract
After contact activation, factor XII can convert factor VII into its active form. Factor XI enhances this action but appears not to be a compulsatory part of the system. The short circuit of the intrinsic system constituted by this pathway may be important in triggering the intrinsic coagulation system. * Present address: Rivadavia Hospital, Buenos Aires, Argentina.This publication has 5 references indexed in Scilit:
- Kinetic Aspects of the Interaction of Blood-Clotting EnzymesThrombosis and Haemostasis, 1967
- The Activation of ProaccelerinBritish Journal of Haematology, 1965
- Haemorrhagic Diathesis as the Result of Severe Deficiency of Plasma Thromboplastin Antecedent (PTA, Factor XI)Thrombosis and Haemostasis, 1964
- Arginine Esterase Activity of the Contact (Hageman) FactorThrombosis and Haemostasis, 1962
- The Mechanism of Platelet Aggregation in Relation to HemostasisThrombosis and Haemostasis, 1962