Potentiation of Smad Transactivation by Jun Proteins during a Combined Treatment with Epidermal Growth Factor and Transforming Growth Factor-β in Rat Hepatocytes: ROLE OF PHOSPHATIDYLINOSITOL 3-KINASE-INDUCED AP-1 ACTIVATION
Open Access
- 30 March 2001
- journal article
- Published by Elsevier
- Vol. 276 (13), 10524-10531
- https://doi.org/10.1074/jbc.m005919200
Abstract
No abstract availableKeywords
This publication has 44 references indexed in Scilit:
- A mechanism of repression of TGFbeta / Smad signaling by oncogenic RasGenes & Development, 1999
- Smad3 and Smad4 cooperate with c-Jun/c-Fos to mediate TGF-β-induced transcriptionNature, 1998
- Determinants of specificity in TGF-β signal transductionGenes & Development, 1998
- Smad2 transduces common signals from receptor serine–threonine and tyrosine kinasesGenes & Development, 1998
- TGF-β SIGNAL TRANSDUCTIONAnnual Review of Biochemistry, 1998
- Mads and Smads in TGFβ signallingCurrent Opinion in Cell Biology, 1998
- Human Smad3 and Smad4 Are Sequence-Specific Transcription ActivatorsMolecular Cell, 1998
- TGF-β signalling from cell membrane to nucleus through SMAD proteinsNature, 1997
- Opposing BMP and EGF signalling pathways converge on the TGF-β family mediator Smad1Nature, 1997
- The TGF-beta superfamily: new members, new receptors, and new genetic tests of function in different organisms.Genes & Development, 1994