Transcriptional Pausing at +62 of the HIV-1 Nascent RNA Modulates Formation of the TAR RNA Structure
- 1 June 1998
- journal article
- Published by Elsevier in Molecular Cell
- Vol. 1 (7), 1033-1042
- https://doi.org/10.1016/s1097-2765(00)80103-3
Abstract
No abstract availableKeywords
This publication has 56 references indexed in Scilit:
- The HIV transactivator TAT binds to the CDK-activating kinase and activates the phosphorylation of the carboxy-terminal domain of RNA polymerase IIGenes & Development, 1997
- Purification of a Tat-associated kinase reveals a TFIIH complex that modulates HIV-1 transcription.The EMBO Journal, 1997
- Multiple interactions stabilize a single paused transcription intermediate in which hairpin to 3′ end spacing distinguishes pause and termination pathwaysJournal of Molecular Biology, 1997
- Increased accommodation of nascent RNA in a product site on RNA polymerase II during arrest.Proceedings of the National Academy of Sciences, 1996
- Regulation of transcriptional elongation by RNA polymerase IICurrent Opinion in Genetics & Development, 1995
- GreA-induced transcript cleavage in transcription complexes containing Escherichia coli RNA polymerase is controlled by multiple factors, including nascent transcript location and structure.Journal of Biological Chemistry, 1994
- Dissection of the his Leader Pause Site by Base Substitution Reveals a Multipartite Signal that Includes a Pause RNA HairpinJournal of Molecular Biology, 1993
- Human immunodeficiency virus type 1 transactivator protein, tat, stimulates transcriptional read-through of distal terminator sequences in vitro.Proceedings of the National Academy of Sciences, 1993
- High Affinity Binding of TAR RNA by the Human Immunodeficiency Virus Type-1 tat Protein Requires Base-pairs in the RNA Stem and Amino Acid Residues Flanking the Basic RegionJournal of Molecular Biology, 1993
- Tat trans-activates the human immunodeficiency virus through a nascent RNA targetCell, 1989