β-Amyloid fibrils induce tau phosphorylation and loss of microtubule binding
- 1 April 1995
- Vol. 14 (4), 879-888
- https://doi.org/10.1016/0896-6273(95)90232-5
Abstract
No abstract availableKeywords
This publication has 53 references indexed in Scilit:
- An important role of heparan sulfate proteoglycan (perlecan) in a model system for the deposition and persistence of fibrillar aβ-amyloid in rat brainNeuron, 1994
- Tau protein and the neurofibrillary pathology of Alzheimer's diseaseTrends in Neurosciences, 1993
- Calcium-destabilizing and neurodegenerative effects of aggregated β-amyloid peptide are attenuated by basic FGFBrain Research, 1993
- Abnormal tau phosphorylation at Ser396 in alzheimer's disease recapitulates development and contributes to reduced microtubule bindingNeuron, 1993
- Release of Excess Amyloid β Protein from a Mutant Amyloid β Protein PrecursorScience, 1993
- Immunological and conformational characterization of a phosphorylated immunodominant epitope on the paired helical filaments found in Alzheimer's diseaseBiochemical and Biophysical Research Communications, 1992
- Action of amyloid beta-protein on protein kinase C activityLife Sciences, 1991
- Localization of the Alz-50 epitope in recombinant human microtubule-associated protein tauNeuroscience Letters, 1991
- Neurotrophic and Neurotoxic Effects of Amyloid β Protein: Reversal by Tachykinin NeuropeptidesScience, 1990
- Microtubular reorganization and dendritic growth response in alzheimer's diseaseAnnals of Neurology, 1989