Morphine promotes apoptosis via TLR2, and this is negatively regulated by β-arrestin 2
- 23 January 2009
- journal article
- research article
- Published by Elsevier in Biochemical and Biophysical Research Communications
- Vol. 378 (4), 857-861
- https://doi.org/10.1016/j.bbrc.2008.12.001
Abstract
No abstract availableKeywords
This publication has 31 references indexed in Scilit:
- Proinflammatory cytokines oppose opioid-induced acute and chronic analgesiaBrain, Behavior, and Immunity, 2008
- Reversal of opiate-induced apoptosis by human recombinant growth hormone in murine foetus primary hippocampal neuronal cell culturesProceedings of the National Academy of Sciences, 2008
- Pivotal role for neuronal Toll-like receptors in ischemic brain injury and functional deficitsProceedings of the National Academy of Sciences, 2007
- β-Arrestins 1 and 2 differentially regulate LPS-induced signaling and pro-inflammatory gene expressionMolecular Immunology, 2007
- Toll-like receptor 2 signaling is a mediator of apoptosis in herpes simplex virus-infected microgliaJournal of Neuroinflammation, 2007
- Knockout of the mu opioid receptor enhances the survival of adult-generated hippocampal granule cell neuronsNeuroscience, 2007
- Nuclear Factor κB Signaling in Opioid Functions and Receptor Gene ExpressionJournal of Neuroimmune Pharmacology, 2006
- Overexpression of TLR2 and TLR4 susceptibility to serum deprivation-induced apoptosis in CHO cellsBiochemical and Biophysical Research Communications, 2005
- An Akt/β-Arrestin 2/PP2A Signaling Complex Mediates Dopaminergic Neurotransmission and BehaviorCell, 2005
- Identification of β-Arrestin2 as a G Protein-Coupled Receptor-Stimulated Regulator of NF-κB PathwaysMolecular Cell, 2004