IL-1β, IFN-γ and TNF-α increase vulnerability of pancreatic beta cells to autoimmune destruction
- 7 May 2003
- journal article
- Published by Elsevier in Journal of Autoimmunity
- Vol. 20 (4), 303-312
- https://doi.org/10.1016/s0896-8411(03)00039-8
Abstract
No abstract availableKeywords
This publication has 46 references indexed in Scilit:
- Dominant Role of Intercellular Adhesion Molecule-1 in the Pathogenesis of Autoimmune Diabetes in Non-obese Diabetic MiceJournal of Autoimmunity, 2001
- The Immunological Synapse: A Molecular Machine Controlling T Cell ActivationScience, 1999
- Dancing the Immunological Two-StepScience, 1999
- CO-CULTURE OF PANCREATIC ISLETS AND ALLOGENEIC LYMPHOCYTESTransplantation, 1997
- Fas and Perforin Pathways as Major Mechanisms of T Cell-Mediated CytotoxicityScience, 1994
- Technical tipsTrends in Genetics, 1994
- Sialic acids as important molecules in the regulation of the immune system: pathophysiological implications of sialidases in immunityGlycobiology, 1993
- Induction of intercellular adhesion molecule-1 (CD54) on isolated mouse pancreatic β cells by inflammatory cytokinesClinical Immunology and Immunopathology, 1992
- Measurement of Insulin in Human Sera Using a New RIA Kit. 1. Insulin Determination in the Absence of Insulin Antibodies — Conventional Assay and Micro Modification2)Experimental and Clinical Endocrinology & Diabetes, 1987
- Cleavage of Structural Proteins during the Assembly of the Head of Bacteriophage T4Nature, 1970