Itch−/−αβ and γδ T cells independently contribute to autoimmunity in Itchy mice
Open Access
- 15 April 2008
- journal article
- Published by American Society of Hematology in Blood
- Vol. 111 (8), 4273-7282
- https://doi.org/10.1182/blood-2007-10-115667
Abstract
E3 ubiquitin ligases determine which intracellular proteins are targets of the ubiquitin conjugation pathway and thus play a key role in determiningKeywords
This publication has 54 references indexed in Scilit:
- Unique Maturation Program of the IgE Response In VivoImmunity, 2007
- Ndfip1 Protein Promotes the Function of Itch Ubiquitin Ligase to Prevent T Cell Activation and T Helper 2 Cell-Mediated InflammationImmunity, 2006
- Phenotypically distinct B cell development pathways map to the three B cell lineages in the mouseProceedings of the National Academy of Sciences, 2006
- B cell clones that sustain long-term plasmablast growth in T-independent extrafollicular antibody responsesProceedings of the National Academy of Sciences, 2006
- Ubiquitin Ligases and the Immune ResponseAnnual Review of Immunology, 2004
- The HECT Domain Ligase Itch Ubiquitinates Endophilin and Localizes to the trans-Golgi Network and Endosomal SystemJournal of Biological Chemistry, 2004
- Signals for Sorting of Transmembrane Proteins to Endosomes and LysosomesAnnual Review of Biochemistry, 2003
- Notch–RBP-J signaling is involved in cell fate determination of marginal zone B cellsNature Immunology, 2002
- Interleukin 9–induced In Vivo Expansion of the B-1 Lymphocyte PopulationThe Journal of Experimental Medicine, 1999
- UBE3A/E6-AP mutations cause Angelman syndromeNature Genetics, 1997