Paraballism caused by bilateral hemorrhagic infarction in basal ganglia
- 1 April 1981
- journal article
- research article
- Published by Wolters Kluwer Health in Neurology
- Vol. 31 (4_part_2), 484-486
- https://doi.org/10.1212/wnl.31.4_part_2.484
Abstract
Bilatera1 ballism developed gradually in a 75-yearold woman after head trauma. Computerized tomography revealed bilateral hemorrhagic infarction in the head of the caudate nucleus and the putamen. Haloperidol abolished the symptoms completely within 2 days. After 18 days, haloperidol was gradually withdrawn over 16 days. The patient remained without ballistic movements thereafter.This publication has 4 references indexed in Scilit:
- Treatment of vascular hemiballism and hemichoreaNeurology, 1977
- Treatment and Prognosis of HemiballismusNew England Journal of Medicine, 1976
- TRANSIENT HEMIBALLISMAnnals of Internal Medicine, 1960
- BALLISM AND THE SUBTHALAMIC NUCLEUS (NUCLEUS HYPOTHALAMICUS; CORPUS LUYSI)Archives of Neurology & Psychiatry, 1947