Hepatocyte Necrosis Induced by Oxidative Stress and IL-1α Release Mediate Carcinogen-Induced Compensatory Proliferation and Liver Tumorigenesis
Top Cited Papers
- 1 August 2008
- journal article
- research article
- Published by Elsevier in Cancer Cell
- Vol. 14 (2), 156-165
- https://doi.org/10.1016/j.ccr.2008.06.016
Abstract
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This publication has 56 references indexed in Scilit:
- NF-κB Is a Negative Regulator of IL-1β Secretion as Revealed by Genetic and Pharmacological Inhibition of IKKβCell, 2007
- p38α suppresses normal and cancer cell proliferation by antagonizing the JNK–c-Jun pathwayNature Genetics, 2007
- Loss of hepatic NF-κB activity enhances chemical hepatocarcinogenesis through sustained c-Jun N-terminal kinase 1 activationProceedings of the National Academy of Sciences, 2006
- Novel Aspects of Oxidative Stress-Associated CarcinogenesisAntioxidants and Redox Signaling, 2006
- Pathogen Recognition and Innate ImmunityCell, 2006
- Attenuated phosphorylation of heat shock protein 27 correlates with tumor progression in patients with hepatocellular carcinomaBiochemical and Biophysical Research Communications, 2005
- AP-1 as a regulator of cell life and deathNature Cell Biology, 2002
- Bcl-2 Expression Inhibits Liver Carcinogenesis and Delays the Development of Proliferating FociThe American Journal of Pathology, 2002
- NFkappaB prevents apoptosis and liver dysfunction during liver regeneration.Journal of Clinical Investigation, 1998
- Tissue-specific regulation of glutamine synthetase gene expression in acute pancreatitis is confirmed by using interleukin-1 receptor knockout miceSurgery, 1996