Epithelial-to-Mesenchymal Transition Is a Potential Pathway Leading to Podocyte Dysfunction and Proteinuria
Top Cited Papers
- 1 February 2008
- journal article
- Published by Elsevier in The American Journal of Pathology
- Vol. 172 (2), 299-308
- https://doi.org/10.2353/ajpath.2008.070057
Abstract
No abstract availableKeywords
Funding Information
- National Institutes of Health (DK071040, DK061408, DK064005)
This publication has 58 references indexed in Scilit:
- Proteinuria precedes podocyte abnormalities inLamb2-/- mice, implicating the glomerular basement membrane as an albumin barrierJournal of Clinical Investigation, 2006
- Epithelial Origin of Myofibroblasts during Fibrosis in the LungProceedings of the American Thoracic Society, 2006
- The podocyte's response to injury: Role in proteinuria and glomerulosclerosisKidney International, 2006
- Hepatocyte Growth Factor Attenuates Liver Fibrosis Induced by Bile Duct LigationThe American Journal of Pathology, 2006
- Podocyte-Specific Deletion of Integrin-Linked Kinase Results in Severe Glomerular Basement Membrane Alterations and Progressive GlomerulosclerosisJournal of the American Society of Nephrology, 2006
- Upregulation of nestin, vimentin, and desmin in rat podocytes in response to injuryVirchows Archiv, 2006
- Pathophysiologic Implications of Reduced Podocyte Number in a Rat Model of Progressive Glomerular InjuryThe American Journal of Pathology, 2006
- Early Glomerular Filtration Defect and Severe Renal Disease in Podocin-Deficient MiceMolecular and Cellular Biology, 2004
- Podocin, a raft-associated component of the glomerular slit diaphragm, interacts with CD2AP and nephrinJournal of Clinical Investigation, 2001
- The transcription factor Snail is a repressor of E-cadherin gene expression in epithelial tumour cellsNature Cell Biology, 2000