Opposing Effects of Protein Kinase C δ and Protein Kinase B α on H2O2-Induced Apoptosis in CHO Cells
- 2 November 1999
- journal article
- Published by Elsevier in Biochemical and Biophysical Research Communications
- Vol. 264 (3), 840-846
- https://doi.org/10.1006/bbrc.1999.1579
Abstract
No abstract availableKeywords
This publication has 30 references indexed in Scilit:
- The Pleckstrin Homology Domain of Protein Kinase D Interacts Preferentially with the η Isoform of Protein Kinase CJournal of Biological Chemistry, 1999
- Protein Kinase Cζ Is a Negative Regulator of Protein Kinase B ActivityJournal of Biological Chemistry, 1999
- Activation of protein kinase C δ by the c-Abl tyrosine kinase in response to ionizing radiationOncogene, 1998
- The Proteolytic Cleavage of Protein Kinase C Isotypes, Which Generates Kinase and Regulatory Fragments, Correlates with Fas‐Mediated and 12‐O‐Tetradecanoyl‐Phorbol‐13‐Acetate‐Induced ApoptosisEuropean Journal of Biochemistry, 1997
- Interleukin-3-Induced Phosphorylation of BAD Through the Protein Kinase AktScience, 1997
- Akt Phosphorylation of BAD Couples Survival Signals to the Cell-Intrinsic Death MachineryCell, 1997
- Protein Kinase C δ Inhibits the Proliferation of Vascular Smooth Muscle Cells by Suppressing G1 Cyclin ExpressionPublished by Elsevier ,1997
- Activation of a CrmA-insensitive, p35-sensitive Pathway in Ionizing Radiation-induced ApoptosisPublished by Elsevier ,1997
- The role of intracellular oxidants in apoptosisBiochimica et Biophysica Acta (BBA) - Molecular Basis of Disease, 1995
- Reactive oxygen species and the regulation of cell death by the Bcl-2 gene familyBiochimica et Biophysica Acta (BBA) - Molecular Basis of Disease, 1995