Inactivation of the p53 gene by either mutation or HPV infection is extremely frequent in human oral squamous cell carcinoma cell lines
- 1 January 1994
- journal article
- Published by Elsevier in Oral Oncology
- Vol. 30 (5), 338-345
- https://doi.org/10.1016/0964-1955(94)90036-1
Abstract
No abstract availableKeywords
This publication has 22 references indexed in Scilit:
- p53 Mutations in Human CancersScience, 1991
- Identification of p53 Gene Mutations in Bladder Cancers and Urine SamplesScience, 1991
- Mutational hot spot in the p53 gene in human hepatocellular carcinomasNature, 1991
- p53: A Frequent Target for Genetic Abnormalities in Lung CancerScience, 1989
- High incidence of lung, bone, and lymphoid tumors in transgenic mice overexpressing mutant alleles of the p53 oncogene.Molecular and Cellular Biology, 1989
- Deletion of 5'-coding sequences of the cellular p53 gene in mouse erythroleukemia: a novel mechanism of oncogene regulation.Molecular and Cellular Biology, 1987
- The cellular oncogene p53 can be activated by mutagenesisNature, 1985
- Overproduction of p53 antigen makes established cells highly tumorigenicNature, 1985
- Structure and transcription of human papillomavirus sequences in cervical carcinoma cellsNature, 1985
- Participation of p53 cellular tumour antigen in transformation of normal embryonic cellsNature, 1984