Scleroderma Fibroblasts Demonstrate Enhanced Activation of Akt (Protein Kinase B) In Situ
- 1 February 2005
- journal article
- Published by Elsevier in Journal of Investigative Dermatology
- Vol. 124 (2), 298-303
- https://doi.org/10.1111/j.0022-202x.2004.23559.x
Abstract
No abstract availableKeywords
This publication has 43 references indexed in Scilit:
- Transforming growth factor-β and connective tissue growth factor: key cytokines in scleroderma pathogenesisCurrent Opinion in Rheumatology, 2001
- A Positive Regulatory Role for Cbl Family Proteins in Tumor Necrosis Factor-related Activation-induced Cytokine (TRANCE) and CD40L-mediated Akt ActivationJournal of Biological Chemistry, 2001
- Phosphatidylinositol 3-Kinase Function Is Required for Transforming Growth Factor β-mediated Epithelial to Mesenchymal Transition and Cell MigrationJournal of Biological Chemistry, 2000
- Phosphatidylinositol 3-Kinase and NF-κB/Rel Are at the Divergence of CD40-Mediated Proliferation and Survival PathwaysThe Journal of Immunology, 2000
- Cellular survival: a play in three AktsGenes & Development, 1999
- Akt Promotes Cell Survival by Phosphorylating and Inhibiting a Forkhead Transcription FactorCell, 1999
- TGFβ—a role in systemic sclerosis?The Journal of Pathology, 1998
- Interleukin-3-Induced Phosphorylation of BAD Through the Protein Kinase AktScience, 1997
- Transforming growth factor-beta 1 induces alpha-smooth muscle actin expression in granulation tissue myofibroblasts and in quiescent and growing cultured fibroblastsThe Journal of cell biology, 1993
- Fibroblast selection in sclerodermaArthritis & Rheumatism, 1982