Toll-like Receptor-2 Is Essential for the Development of Palmitate-induced Insulin Resistance in Myotubes
Open Access
- 1 September 2006
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 281 (37), 26865-26875
- https://doi.org/10.1074/jbc.m513304200
Abstract
No abstract availableKeywords
This publication has 53 references indexed in Scilit:
- Palmitate Activates the NF-κB Transcription Factor and Induces IL-6 and TNFα Expression in 3T3-L1 AdipocytesJournal of Nutrition, 2005
- Palmitate-Induced Interleukin 6 Production Is Mediated by Protein Kinase C and Nuclear-Factor κB Activation and Leads to Glucose Transporter 4 Down-Regulation in Skeletal Muscle CellsEndocrinology, 2005
- Circulating Mononuclear Cells in the Obese Are in a Proinflammatory StateCirculation, 2004
- Medical Consequences of ObesityJournal of Clinical Endocrinology & Metabolism, 2004
- Saturated Fatty Acid Activates but Polyunsaturated Fatty Acid Inhibits Toll-like Receptor 2 Dimerized with Toll-like Receptor 6 or 1Journal of Biological Chemistry, 2004
- Cellular mechanism of insulin resistance: potential links with inflammationInternational Journal of Obesity, 2003
- Effects of Free Fatty Acids (FFA) on Glucose Metabolism: Significance for Insulin Resistance and Type 2 DiabetesExperimental and Clinical Endocrinology & Diabetes, 2003
- Differential modulation of Toll-like receptors by fatty acids: preferential inhibition by n-3 polyunsaturated fatty acidsJournal of Lipid Research, 2003
- Oxidative Stress and Stress-Activated Signaling Pathways: A Unifying Hypothesis of Type 2 DiabetesEndocrine Reviews, 2002
- Early events involved in the development of insulin resistance in Zucker fatty ratInternational Journal of Obesity, 2002