Role of interleukin‐1β and tumour necrosis factor‐α in lipopolysaccharide‐induced sickness behaviour: a study with interleukin‐1 type I receptor‐deficient mice
- 1 December 2000
- journal article
- Published by Wiley in European Journal of Neuroscience
- Vol. 12 (12), 4447-4456
- https://doi.org/10.1111/j.1460-9568.2000.01348.x
Abstract
No abstract availableKeywords
This publication has 47 references indexed in Scilit:
- Brain mechanisms in cytokine-induced anorexiaPsychoneuroendocrinology, 1999
- The Interleukin-1 Receptor Accessory Protein (IL-1RAcP) Is Essential for IL-1-induced Activation of Interleukin-1 Receptor-associated Kinase (IRAK) and Stress-activated Protein Kinases (SAP Kinases)Journal of Biological Chemistry, 1997
- Central mediation of the effects of interleukin-1 on social exploration and body weight in micePsychoneuroendocrinology, 1997
- Inhibition of tumor necrosis factor is protective against neurologic dysfunction after active immunization of lewis rats with myelin basic proteinExperimental Neurology, 1995
- The type II ‘decoy’ receptor: A novel regulatory pathway for interleukin 1Immunology Today, 1994
- Synergy between tumor necrosis factor α and interleukin-1 in the induction of sickness behavior in micePsychoneuroendocrinology, 1994
- Type I interleukin-1 receptors in the mouse brain-endocrine-immune axis labelled with [125I]recombinant human interleukin-1 receptor antagonistJournal of Neuroimmunology, 1992
- Intracerebroventricular administration of a specific IL-1 receptor antagonist blocks food and water intake suppression induced by interleukin-1βPhysiology & Behavior, 1992
- Sickness behavior as a new target for drug developmentTrends in Pharmacological Sciences, 1992
- cDNA Expression Cloning of the IL-1 Receptor, a Member of the Immunoglobulin SuperfamilyScience, 1988