Promising molecular targets for cancer prevention: AP-1, NF-κB and Pdcd4
- 1 January 2003
- journal article
- review article
- Published by Elsevier in Trends in Molecular Medicine
- Vol. 9 (1), 36-41
- https://doi.org/10.1016/s1471-4914(02)00009-6
Abstract
No abstract availableKeywords
This publication has 49 references indexed in Scilit:
- Up-regulation of PDCD4 in senescent human diploid fibroblastsBiochemical and Biophysical Research Communications, 2002
- NF-κB in cancer: from innocent bystander to major culpritNature Reviews Cancer, 2002
- Transactivation of Fra-1 and Consequent Activation of AP-1 Occur Extracellular Signal-Regulated Kinase DependentlyMolecular and Cellular Biology, 2002
- Detection of differentially expressed genes in human colon carcinoma cells treated with a selective COX-2 inhibitorOncogene, 2001
- AP-1 in mouse development and tumorigenesisOncogene, 2001
- A novel transformation suppressor, Pdcd4, inhibits AP-1 transactivation but not NF-κB or ODC transactivationOncogene, 2001
- Induced expression of dominant-negative c-jun downregulates NF?B and AP-1 target genes and suppresses tumor phenotype in human keratinocytesMolecular Carcinogenesis, 2000
- Neoplastic transformation of rat thyroid cells requires the junB and fra-1 gene induction which is dependent on the HMGI-C gene productThe EMBO Journal, 1997
- Transformation by ras modifies AP1 composition and activityOncogene, 1997
- Isolation of a novel mouse gene MA-3 that is induced upon programmed cell deathGene, 1995