Long-Term Stress Degenerates, But Imipramine Regenerates, Noradrenergic Axons in the Rat Cerebral Cortex
- 1 October 1997
- journal article
- Published by Elsevier in Biological Psychiatry
- Vol. 42 (8), 687-696
- https://doi.org/10.1016/s0006-3223(96)00502-1
Abstract
No abstract availableKeywords
Funding Information
- Ministry of Education, Culture, Sports, Science and Technology
This publication has 23 references indexed in Scilit:
- Degeneration of locus coeruleus axons in stress-induced depression modelBrain Research Bulletin, 1994
- Quantitative Cerebral Anatomy in DepressionArchives of General Psychiatry, 1993
- Subcortical hyperintensity on magnetic resonance imaging: a comparison of normal and depressed elderly subjectsAmerican Journal of Psychiatry, 1990
- Subcortical Abnormalities Detected in Bipolar Affective Disorder Using Magnetic Resonance ImagingArchives of General Psychiatry, 1990
- Quantitation of catecholamine neurons in the locus coeruleus in human brains of normal young and older adults and in depressionJournal of Comparative Neurology, 1989
- Chronic immobilization stress: evidence for decreases of 5-hydroxy-tryptamine immunoreactivity and for increases of glucocorticoid receptor immunoreactivity in various brain regions of the male ratJournal of Neural Transmission, 1989
- Effects of chronic imipramine treatment on glucocorticoid receptor immunoreactivity in various regions of the rat brainJournal of Neural Transmission, 1988
- Psychoendocrine model of depressionPsychoneuroendocrinology, 1979
- Clinical and experimental studies on the pathogenesis of depressionPsychoneuroendocrinology, 1977
- Development of β-adrenergic receptor subsensitivity by antidepressantsNature, 1977