Nitrotyrosine and protein carbonyls are equally distributed in HT22 cells after nitrosative stress
- 1 March 2007
- journal article
- Published by Elsevier in Free Radical Biology & Medicine
- Vol. 42 (6), 773-786
- https://doi.org/10.1016/j.freeradbiomed.2006.11.029
Abstract
No abstract availableThis publication has 117 references indexed in Scilit:
- Intracellular distribution of oxidized proteins and proteasome in HT22 cells during oxidative stressFree Radical Biology & Medicine, 2006
- Mitochondria in homeostasis of reactive oxygen species in cell, tissues, and organismThe International Journal of Biochemistry & Cell Biology, 2005
- Reactive oxygen and nitrogen species: Friends or foes?Biochemistry (Moscow), 2005
- Consumption of nitric oxide by endothelial cells: Evidence for the involvement of a NAD(P)H‐, flavin‐ and heme‐dependent dioxygenase reactionFEBS Letters, 2004
- Hypoxia Potentiates Nitric Oxide-mediated Apoptosis in Endothelial Cells via Peroxynitrite-induced Activation of Mitochondria-dependent and -independent PathwaysPublished by Elsevier ,2004
- Endothelial cell activation by endotoxin involves superoxide/NO‐mediated nitration of prostacyclin synthase and thromboxane receptor stimulationThe FASEB Journal, 2003
- PARP‐mediated proteasome activation: A co‐ordination of DNA repair and protein degradation?BioEssays, 2002
- Proteasome inhibition by lipofuscin/ceroid during postmitotic aging of fibroblastsThe FASEB Journal, 2000
- Oxidative Damage and Tyrosine Nitration from PeroxynitriteChemical Research in Toxicology, 1996
- Peroxynitrite‐mediated oxidative protein modificationsFEBS Letters, 1995