Frontotemporal dementia, chromosome 17, and progranulin
- 18 September 2006
- journal article
- editorial
- Published by Wiley in Annals of Neurology
- Vol. 60 (3), 275-277
- https://doi.org/10.1002/ana.20962
Abstract
No abstract availableThis publication has 32 references indexed in Scilit:
- HDDD2 is a familial frontotemporal lobar degeneration with ubiquitin‐positive, tau‐negative inclusions caused by a missense mutation in the signal peptide of progranulinAnnals of Neurology, 2006
- Mutations in progranulin cause tau-negative frontotemporal dementia linked to chromosome 17Nature, 2006
- Null mutations in progranulin cause ubiquitin-positive frontotemporal dementia linked to chromosome 17q21Nature, 2006
- Frontotemporal dementiaThe Lancet Neurology, 2005
- Primary lateral sclerosis, hereditary spastic paraplegia, and mutations in thealsingene: Historical background for the first International ConferenceAmyotrophic Lateral Sclerosis, 2005
- A clinical pathological comparison of three families with frontotemporal dementia and identical mutations in the tau gene (P301L)Brain, 1999
- Association of missense and 5′-splice-site mutations in tau with the inherited dementia FTDP-17Nature, 1998
- Frontotemporal dementia and parkinsonism linked to chromosome 17: A consensus conferenceAnnals of Neurology, 1997
- Clinical and neuropathological criteria for frontotemporal dementia. The Lund and Manchester Groups.Journal of Neurology, Neurosurgery & Psychiatry, 1994
- Frontal lobe degeneration of non-Alzheimer type. I. NeuropathologyArchives of Gerontology and Geriatrics, 1987