Anosmin-1, Defective in the X-Linked Form of Kallmann Syndrome, Promotes Axonal Branch Formation from Olfactory Bulb Output Neurons
- 1 April 2002
- Vol. 109 (2), 217-228
- https://doi.org/10.1016/s0092-8674(02)00713-4
Abstract
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This publication has 50 references indexed in Scilit:
- Olfactory SystemPublished by Elsevier ,2004
- Anosmin-1 is a regionally restricted component of basement membranes and interstitial matrices during organogenesis: Implications for the developmental anomalies of X chromosome-linked Kallmann syndromeDevelopmental Dynamics, 1999
- The Kallmann syndrome gene product expressed in COS cells is cleaved on the cell surface to yield a diffusible componentHuman Molecular Genetics, 1996
- Cloning and expression of the mouse pseudoautosomal steroid sulphatase gene (Sts)Nature Genetics, 1996
- Control of Topographic Retinal Axon Branching by Inhibitory Membrane-Bound MoleculesScience, 1994
- Chemorepulsion of axons in the developing mammalian central nervous systemScience, 1993
- The candidate gene for the X-linked Kallmann syndrome encodes a protein related to adhesion moleculesCell, 1991
- A gene deleted in Kallmann's syndrome shares homology with neural cell adhesion and axonal path-finding moleculesNature, 1991
- A diverse set of developmentally regulated proteoglycans is expressed in the rat central nervous systemNeuron, 1990
- Cortical axons branch to multiple subcortical targets by interstitial axon budding: Implications for target recognition and “waiting periods”Neuron, 1988