Differential Activation of Signal Transduction Pathways in Human Hearts With Hypertrophy Versus Advanced Heart Failure
Top Cited Papers
- 6 February 2001
- journal article
- other
- Published by Wolters Kluwer Health in Circulation
- Vol. 103 (5), 670-677
- https://doi.org/10.1161/01.cir.103.5.670
Abstract
Background—Left ventricular failure is commonly preceded by a period of hypertrophy. Intriguingly, many of the signaling pathways that have been implicated in the regulation of hypertrophy, includi...Keywords
This publication has 15 references indexed in Scilit:
- Glycogen Synthase Kinase-3β Is a Negative Regulator of Cardiomyocyte HypertrophyThe Journal of cell biology, 2000
- c-Jun NH2-Terminal Kinase Inhibits Targeting of the Protein Phosphatase Calcineurin to NFATc1Molecular and Cellular Biology, 2000
- Role of the Adenomatous Polyposis Coli Gene Product in Human Cardiac Development and DiseaseJournal of Biological Chemistry, 2000
- Revisiting calcineurin and human heart failureNature Medicine, 2000
- Stress Pathways and Heart FailureCell, 1999
- Activation of c-Jun N-Terminal Kinases and p38-Mitogen-activated Protein Kinases in Human Heart Failure Secondary to Ischaemic Heart DiseaseJournal of Molecular and Cellular Cardiology, 1999
- Regulation of cardiac hypertrophy in vivo by the stress-activated protein kinases/c-Jun NH2-terminal kinasesJournal of Clinical Investigation, 1999
- IGF-1 induces skeletal myocyte hypertrophy through calcineurin in association with GATA-2 and NF-ATc1Nature, 1999
- Apoptosis in heart failure: Release of cytochrome c from mitochondria and activation of caspase-3 in human cardiomyopathyProceedings of the National Academy of Sciences, 1999
- Protein Kinase B/Akt Participates in GLUT4 Translocation by Insulin in L6 MyoblastsMolecular and Cellular Biology, 1999