The carboxyl-terminal of BRCA1 is required for subnuclear assembly of RAD51 after treatment with cisplatin but not ionizing radiation in human breast and ovarian cancer cells
- 28 October 2005
- journal article
- Published by Elsevier in Biochemical and Biophysical Research Communications
- Vol. 336 (3), 952-960
- https://doi.org/10.1016/j.bbrc.2005.08.197
Abstract
No abstract availableKeywords
This publication has 46 references indexed in Scilit:
- The BRCT Domain Is a Phospho-Protein Binding DomainScience, 2003
- Cisplatin: mode of cytotoxic action and molecular basis of resistanceOncogene, 2003
- Nuclease-Deficient FEN-1 Blocks Rad51/BRCA1-Mediated Repair and Causes Trinucleotide Repeat InstabilityMolecular and Cellular Biology, 2003
- Role of BRCA1 in cellular resistance to paclitaxel and ionizing radiation in an ovarian cancer cell line carrying a defective BRCA1Oncogene, 2003
- Biallelic Inactivation of BRCA2 in Fanconi AnemiaScience, 2002
- BRCA1 deficient embryonic stem cells display a decreased homologous recombination frequency and an increased frequency of non-homologous recombination that is corrected by expression of a Brca1 transgeneOncogene, 1999
- Association of BRCA1 with the hRad50-hMre11-p95 Complex and the DNA Damage ResponseScience, 1999
- The Nuclear Localization Sequences of the BRCA1 Protein Interact with the Importin-α Subunit of the Nuclear Transport Signal ReceptorJournal of Biological Chemistry, 1996
- The Tumor Suppressor Gene Brca1 Is Required for Embryonic Cellular Proliferation in the MouseCell, 1996
- Linkage of Early-Onset Familial Breast Cancer to Chromosome 17q21Science, 1990