Role of Ubiquitin Carboxy Terminal Hydrolase-L1 in Neural Cell Apoptosis Induced by Ischemic Retinal Injury in Vivo
- 31 January 2004
- journal article
- research article
- Published by Elsevier in The American Journal of Pathology
- Vol. 164 (1), 59-64
- https://doi.org/10.1016/s0002-9440(10)63096-9
Abstract
No abstract availableKeywords
This publication has 53 references indexed in Scilit:
- Neurotrophic Signaling in Normal and Degenerating Rodent RetinasExperimental Eye Research, 2001
- A hypothesis to explain ganglion cell death caused by vascular insults at the optic nerve head: possible implication for the treatment of glaucomaBritish Journal of Ophthalmology, 2001
- Role of Muller cells in retinal degenerationsFrontiers in Bioscience-Landmark, 2001
- Apoptotic PathwaysCell, 2000
- Ubiquitin Protein Ligase Activity of IAPs and Their Degradation in Proteasomes in Response to Apoptotic StimuliScience, 2000
- BDNF and NT-4 differentially modulate neurite outgrowth in developing retinal ganglion cellsJournal of Neuroscience Research, 1999
- Neurotrophin‐4/5 (NT‐4/5) increases adult rat retinal ganglion cell survival and neurite outgrowth in vitroJournal of Neurobiology, 1994
- Ubiquitin carboxyl‐terminal hydrolase (PGP 9.5) is selectively present in ubiquitinated inclusion bodies characteristic of human neurodegenerative diseasesThe Journal of Pathology, 1990
- The Neuron-Specific Protein PGP 9.5 Is a Ubiquitin Carboxyl-Terminal HydrolaseScience, 1989
- The yeast polyubiquitin gene is essential for resistance to high temperatures, starvation, and other stressesCell, 1987