Differential regulation of protooncogenes c-jun and jun D expressions by protein tyrosine kinase, protein kinase C, and cyclic-AMP mitogenic pathways in dog primary thyrocytes: TSH and cyclic-AMP induce proliferation but downregulate C-jun expression
- 1 October 1991
- journal article
- Published by Elsevier in Experimental Cell Research
- Vol. 196 (2), 210-215
- https://doi.org/10.1016/0014-4827(91)90253-q
Abstract
No abstract availableThis publication has 28 references indexed in Scilit:
- The c-fos protein interacts with c-JunAP-1 to stimulate transcription of AP-1 responsive genesCell, 1988
- fos-associated cellular p39 is related to nuclear transcription factor AP-1Cell, 1988
- Induction of proto-oncogene JUN/AP-1 by serum and TPANature, 1988
- Epidermal growth factor stimulates transcription of the c-jun proto-oncogene in rat fibroblastsNature, 1988
- Oncogene jun encodes a sequence-specific trans- activator similar to AP-1Nature, 1988
- Common DNA binding site for Fos protein complexesand transcription factor AP-1Cell, 1988
- Human Proto-Oncogene c- jun Encodes a DNA Binding Protein with Structural and Functional Properties of Transcription Factor AP-1Science, 1987
- Regulation of dog thyroid epithelial cell cycle by forskolin, an adenylate cyclase activatorExperimental Cell Research, 1987
- Induction of DNA synthesis in dog thyrocytes in primary culture: Synergistic effects of thyrotropin and cyclic AMP with epidermal growth factor and insulinJournal of Cellular Physiology, 1987
- Stimulation by thyrotropin and cyclic AMP of the proliferation of quiescent canine thyroid cells cultured in a defined medium containing insulinFEBS Letters, 1983