Nuclear Inclusions in Glutamine Repeat Disorders
- 1 October 1998
- Vol. 95 (1), 1-4
- https://doi.org/10.1016/s0092-8674(00)81743-2
Abstract
No abstract availableThis publication has 20 references indexed in Scilit:
- Ataxin-1 Nuclear Localization and AggregationCell, 1998
- Polyglutamine-Expanded Human Huntingtin Transgenes Induce Degeneration of Drosophila Photoreceptor NeuronsNeuron, 1998
- Chaperone suppression of aggregation and altered subcellular proteasome localization imply protein misfolding in SCA1Nature Genetics, 1998
- The Influence of Huntingtin Protein Size on Nuclear Localization and Cellular ToxicityThe Journal of cell biology, 1998
- Aggregation of Huntingtin in Neuronal Intranuclear Inclusions and Dystrophic Neurites in BrainScience, 1997
- Formation of Neuronal Intranuclear Inclusions Underlies the Neurological Dysfunction in Mice Transgenic for the HD MutationCell, 1997
- Mad Cows Meet Psi-chotic Yeast: The Expansion of the Prion HypothesisCell, 1997
- A huntingtin-associated protein enriched in brain with implications for pathologyNature, 1995
- SCA1 transgenic mice: A model for neurodegeneration caused by an expanded CAG trinucleotide repeatCell, 1995
- Inactivation of the Mouse Huntington's Disease Gene Homolog HdhScience, 1995