High glucose disrupts oligosaccharide recognition function via competitive inhibition: A potential mechanism for immune dysregulation in diabetes mellitus
- 1 July 2010
- journal article
- research article
- Published by Elsevier in Immunobiology
- Vol. 216 (1-2), 126-131
- https://doi.org/10.1016/j.imbio.2010.06.002
Abstract
No abstract availableKeywords
This publication has 47 references indexed in Scilit:
- Lymphocytes in the Peritoneum Home to the Omentum and Are Activated by Resident Dendritic CellsThe Journal of Immunology, 2009
- Acute Modulation of Toll-Like Receptors by InsulinDiabetes Care, 2008
- The lectin‐like activity of human C1q and its implication in DNA and apoptotic cell recognitionFEBS Letters, 2008
- Increased Toll-Like Receptor (TLR) 2 and TLR4 Expression in Monocytes from Patients with Type 1 Diabetes: Further Evidence of a Proinflammatory StateJournal of Clinical Endocrinology & Metabolism, 2008
- Mannose-binding lectin enhances Toll-like receptors 2 and 6 signaling from the phagosomeThe Journal of Experimental Medicine, 2008
- Metabolic syndrome and obesity in an insectProceedings of the National Academy of Sciences, 2006
- DC-SIGN and CLEC-2 Mediate Human Immunodeficiency Virus Type 1 Capture by PlateletsJournal of Virology, 2006
- Macrophage-secreted factors induce adipocyte inflammation and insulin resistanceBiochemical and Biophysical Research Communications, 2006
- An Endogenous Drosophila Receptor for Glycans Bearing α1,3-Linked Core Fucose ResiduesPublished by Elsevier ,2002
- THE BASAL PLASMA GLUCOSE: A SIMPLE RELEVANT INDEX OF MATURITY‐ONSET DIABETESClinical Endocrinology, 1981