Dynorphin increases in the dorsal spinal cord in rats with a painful peripheral neuropathy
- 1 July 1990
- Vol. 11 (4), 719-728
- https://doi.org/10.1016/0196-9781(90)90187-a
Abstract
No abstract availableKeywords
This publication has 30 references indexed in Scilit:
- Further evidence for ‘pain-related’ behaviours in a model of unilateral peripheral mononeuropathyPain, 1990
- κ-Opioid receptors and analgesiaTrends in Pharmacological Sciences, 1990
- Peripheral inflammation is associated with increased dynorphin immunoreactivity in both projection and local circuit neurons in the superficial dorsal horn of the rat lumbar spinal cordNeuroscience Letters, 1989
- Opioid peptide gene expression in rat trigeminal nucleus caudalis neurons: Normal distribution and effects of trigeminal deafferentationJournal of Comparative Neurology, 1988
- Immunocytochemical identification of long ascending, peptidergic lumbar spinal neurons terminating in either the medial or lateral thalamus in the ratBrain Research, 1988
- Increased staining of immunoreactive dynorphin cell bodies in the deafferented spinal cord of the ratNeuroscience Letters, 1988
- Prodynorphin gene expression is enhanced in the spinal cord of chronic arthritic ratsNeuroscience Letters, 1987
- The somatotopic organization of primary afferent terminals in the superficial laminae of the dorsal horn of the rat spinal cordJournal of Comparative Neurology, 1985
- Deafferentation in animals as a model for the study of pain: an alternative hypothesisBrain Research Reviews, 1984
- Autotomy following peripheral nerve lesions: experimental anesthesia dolorosaPain, 1979