Dithiocarbamates Induce Apoptosis in Thymocytes by Raising the Intracellular Level of Redox-active Copper
Open Access
- 1 November 1995
- journal article
- research article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 270 (44), 26202-26208
- https://doi.org/10.1074/jbc.270.44.26202
Abstract
No abstract availableKeywords
This publication has 43 references indexed in Scilit:
- A role for metals and free radicals in the induction of apoptosis in thymocytesFEBS Letters, 1994
- Formation of 50 kbp chromatin fragments in isolated liver nuclei is mediated by protease and endonuclease activationFEBS Letters, 1994
- Bcl‐2 protects from oxidative damage and apoptotic cell death without interfering with activation of NF‐κB by TNFFEBS Letters, 1994
- Inactivation of intracellular copper-zinc superoxide dismutase by copper chelating agents without glutathione depletion and methemoglobin formationFree Radical Biology & Medicine, 1989
- Antioxidant capacity of diethyldithiocarbamate in a metal independent lipid peroxidative processFree Radical Biology & Medicine, 1989
- Optimal dithiocarbamate structure for immunomodulator actionMedical Hypotheses, 1988
- RANDOMISED, DOUBLE-BLIND, PLACEBO-CONTROLLED TRIAL OF DITIOCARB SODIUM ('IMUTHIOL') IN HUMAN IMMUNODEFICIENCY VIRUS INFECTIONThe Lancet, 1988
- Antioxidant effect of diethyldithiocarbamate on microsomal lipid peroxidation assessed by low-level chemiluminescence and alkane productionFEBS Letters, 1983
- Bathocuproine sulphonate: a tissue culture-compatible indicator of copper-mediated toxicityNature, 1983
- A Critical Review of the Literature on Carbon Disulfide ToxicityCRC Critical Reviews in Toxicology, 1983