Mechanisms to control rereplication and implications for cancer
- 28 November 2007
- journal article
- review article
- Published by Elsevier in Current Opinion in Cell Biology
- Vol. 19 (6), 663-671
- https://doi.org/10.1016/j.ceb.2007.10.007
Abstract
No abstract availableKeywords
This publication has 58 references indexed in Scilit:
- Oncogene-induced senescence is a DNA damage response triggered by DNA hyper-replicationNature, 2006
- Oncogene-induced senescence is part of the tumorigenesis barrier imposed by DNA damage checkpointsNature, 2006
- Deregulated Replication Licensing Causes DNA Fragmentation Consistent with Head-to-Tail Fork CollisionMolecular Cell, 2006
- CUL4–DDB1 ubiquitin ligase interacts with multiple WD40-repeat proteins and regulates histone methylationNature Cell Biology, 2006
- DNA damage induces Cdt1 proteolysis in fission yeast through a pathway dependent on Cdt2 and Ddb1EMBO Reports, 2006
- Molecular architecture and assembly of the DDB1–CUL4A ubiquitin ligase machineryNature, 2006
- Live-Cell Imaging Reveals Replication of Individual Replicons in Eukaryotic Replication FactoriesCell, 2006
- Cleavage of Cdc6 by caspase-3 promotes ATM/ATR kinase–mediated apoptosis of HeLa cellsThe Journal of cell biology, 2006
- Contribution of DNA repair and cell cycle checkpoint arrest to the maintenance of genomic stabilityDNA Repair, 2006
- Sequential ATP Hydrolysis by Cdc6 and ORC Directs Loading of the Mcm2-7 HelicaseMolecular Cell, 2006