Inhibition of Alzheimer's disease β‐amyloid aggregation, neurotoxicity, and in vivo deposition by nitrophenols: implications for Alzheimer's therapy
Open Access
- 20 March 2001
- journal article
- fj express-summary
- Published by Wiley in The FASEB Journal
- Vol. 15 (7), 1297-1299
- https://doi.org/10.1096/fj.00-0676fje
Abstract
No abstract availableKeywords
This publication has 41 references indexed in Scilit:
- Properties of cytotoxic peptide-formed ion channelsAmerican Journal of Physiology-Cell Physiology, 2000
- Alzheimer's β-Amyloid, Human Islet Amylin, and Prion Protein Fragment Evoke Intracellular Free Calcium Elevations by a Common Mechanism in a Hypothalamic GnRH Neuronal Cell LineJournal of Biological Chemistry, 2000
- The Presenilins in Alzheimer's Disease--Proteolysis Holds the KeyScience, 1999
- Mechanisms of Neuronal Degeneration in Alzheimer's DiseaseNeuron, 1996
- Zn2+ interaction with Alzheimer amyloid beta protein calcium channels.Proceedings of the National Academy of Sciences, 1996
- Alzheimer's Disease: A Central Role for AmyloidJournal of Neuropathology and Experimental Neurology, 1994
- Giant multilevel cation channels formed by Alzheimer disease amyloid beta-protein [A beta P-(1-40)] in bilayer membranes.Proceedings of the National Academy of Sciences, 1993
- Alzheimer disease amyloid beta protein forms calcium channels in bilayer membranes: blockade by tromethamine and aluminum.Proceedings of the National Academy of Sciences, 1993
- Amyloid plaque core protein in Alzheimer disease and Down syndrome.Proceedings of the National Academy of Sciences, 1985
- Alzheimer's disease: Initial report of the purification and characterization of a novel cerebrovascular amyloid proteinBiochemical and Biophysical Research Communications, 1984