Smooth muscle cell expression of a constitutive active form of human Rac 1 accelerates cutaneous wound repair
- 1 January 2005
- Vol. 137 (1), 92-101
- https://doi.org/10.1016/j.surg.2004.06.012
Abstract
No abstract availableKeywords
This publication has 35 references indexed in Scilit:
- Hypoxia modulates the effects of transforming growth factor-β isoforms on matrix-formation by primary human lung fibroblastsCytokine, 2003
- Plant Rac Proteins Induce Superoxide Production in Mammalian CellsBiochemical and Biophysical Research Communications, 2000
- The Actin Cytoskeleton Reorganization Induced by Rac1 Requires the Production of SuperoxideAntioxidants and Redox Signaling, 1999
- Hypoxia Response Elements in the Aldolase A, Enolase 1, and Lactate Dehydrogenase A Gene Promoters Contain Essential Binding Sites for Hypoxia-inducible Factor 1Journal of Biological Chemistry, 1996
- Plasticity of Vascular Smooth Muscle α-Actin Gene TranscriptionJournal of Biological Chemistry, 1995
- Small GTP-Binding Proteins and the Regulation of the Actin CytoskeletonAnnual Review of Cell Biology, 1994
- The small GTP-binding protein rac regulates growth factor-induced membrane rufflingCell, 1992
- Growth factors in wound healing. Single and synergistic effects on partial thickness porcine skin wounds.Journal of Clinical Investigation, 1989
- Cutaneous tissue repair: Basic biologic considerations. IJournal of the American Academy of Dermatology, 1985
- Blood Vessel Fibronectin Increases in Conjunction with Endothelial Cell Proliferation and Capillary Ingrowth During Wound HealingJournal of Investigative Dermatology, 1982