In vivo interactions of apoA-II, apoA-I, and hepatic lipase contributing to HDL structure and antiatherogenic functions
Open Access
- 1 April 2001
- journal article
- Published by Elsevier in Journal of Lipid Research
- Vol. 42 (4), 563-570
- https://doi.org/10.1016/s0022-2275(20)31165-2
Abstract
No abstract availableKeywords
This publication has 28 references indexed in Scilit:
- Overexpression of apolipoprotein AII in transgenic mice converts high density lipoproteins to proinflammatory particles.Journal of Clinical Investigation, 1997
- Effect of platelet activating factor-acetylhydrolase on the formation and action of minimally oxidized low density lipoprotein.Journal of Clinical Investigation, 1995
- Human ApoA-II inhibits the hydrolysis of HDL triglyceride and the decrease of HDL size induced by hypertriglyceridemia and cholesteryl ester transfer protein in transgenic mice.Journal of Clinical Investigation, 1994
- Atherosclerosis in Transgenic Mice Overexpressing Apolipoprotein A-IIScience, 1993
- Inhibition of early atherogenesis in transgenic mice by human apolipoprotein AINature, 1991
- Expression of human apolipoprotein A-I in transgenic mice results in reduced plasma levels of murine apolipoprotein A-I and the appearance of two new high density lipoprotein size subclasses.Proceedings of the National Academy of Sciences, 1991
- Plasma high density lipoproteins. Metabolism and relationship to atherogenesis.Journal of Clinical Investigation, 1990
- Regression of atherosclerotic lesions by high density lipoprotein plasma fraction in the cholesterol-fed rabbit.Journal of Clinical Investigation, 1990
- Associations of high-density lipoprotein subclasses and apolipoproteins with ischemic heart disease and coronary atherosclerosisAmerican Heart Journal, 1987
- Cholesterol efflux from cultured adipose cells is mediated by LpAI particles but not by LpAI:AII particlesBiochemical and Biophysical Research Communications, 1987