Chronic Nicotine Treatment Up-regulates Human α3β2 but Not α3β4 Acetylcholine Receptors Stably Transfected in Human Embryonic Kidney Cells
Open Access
- 1 November 1998
- journal article
- Published by Elsevier
- Vol. 273 (44), 28721-28732
- https://doi.org/10.1074/jbc.273.44.28721
Abstract
No abstract availableKeywords
This publication has 44 references indexed in Scilit:
- Differential Inhibition by α‐Conotoxin‐MII of the Nicotinic Stimulation of [3H]Dopamine Release from Rat Striatal Synaptosomes and SlicesJournal of Neurochemistry, 1998
- Broad-Spectrum, Non-Opioid Analgesic Activity by Selective Modulation of Neuronal Nicotinic Acetylcholine ReceptorsScience, 1998
- Neuronal Nicotinic Acetylcholine Receptors on Bovine Chromaffin Cells: Cloning, Expression, and Genomic Organization of Receptor SubunitsJournal of Neurochemistry, 1997
- Stable expression in HEK‐293 cells of the rat α3/β4 subtype of neuronal nicotinic acetylcholine receptorFEBS Letters, 1996
- Nicotinic Receptors in the Development and Modulation of CNS SynapsesNeuron, 1996
- Molecular and Cellular Aspects of Nicotine AbuseNeuron, 1996
- Stable expression and pharmacological properties of the human α7 nicotinic acetylcholine receptorEuropean Journal of Pharmacology: Molecular Pharmacology, 1995
- Regions of beta 2 and beta 4 responsible for differences between the steady state dose-response relationships of the alpha 3 beta 2 and alpha 3 beta 4 neuronal nicotinic receptors.The Journal of general physiology, 1995
- Membrane Clustering and Bungarotoxin Binding by the Nicotinic Acetylcholine Receptor: Role of the β SubunitJournal of Neurochemistry, 1994
- Interaction of the 43 kd postsynaptic protein with all subunits of the muscle nicotinic acetylcholine receptorNeuron, 1993