Role of apoE/Aβ interactions in Alzheimer's disease: insights from transgenic mouse models
- 1 February 2002
- journal article
- review article
- Published by Springer Nature in Molecular Psychiatry
- Vol. 7 (2), 132-135
- https://doi.org/10.1038/sj.mp.4001006
Abstract
No abstract availableKeywords
This publication has 14 references indexed in Scilit:
- Apolipoprotein E facilitates neuritic and cerebrovascular plaque formation in an Alzheimer's disease modelAnnals of Neurology, 2000
- Expression of human apolipoprotein E reduces amyloid-β deposition in a mouse model of Alzheimer's diseaseJournal of Clinical Investigation, 1999
- Biology of Aβ Amyloid in Alzheimer's DiseaseNeurobiology of Disease, 1997
- Lack of apolipoprotein E dramatically reduces amyloid β-peptide depositionNature Genetics, 1997
- Correlative Memory Deficits, Aβ Elevation, and Amyloid Plaques in Transgenic MiceScience, 1996
- Apolipoprotein E and Alzheimer's DiseaseAnnual Review of Neuroscience, 1996
- Alzheimer-type neuropathology in transgenic mice overexpressing V717F β-amyloid precursor proteinNature, 1995
- Gating of the voltage-dependent chloride channel CIC-0 by the permeant anionNature, 1995
- Apolipoprotein E in sporadic Alzheimer's disease: Allelic variation and receptor interactionsNeuron, 1993
- Apolipoprotein E: Cholesterol Transport Protein with Expanding Role in Cell BiologyScience, 1988