LPS priming potentiates and prolongs proinflammatory cytokine response to the trichothecene deoxynivalenol in the mouse
- 11 July 2005
- journal article
- research article
- Published by Elsevier in Toxicology and Applied Pharmacology
- Vol. 211 (1), 53-63
- https://doi.org/10.1016/j.taap.2005.04.031
Abstract
No abstract availableKeywords
This publication has 47 references indexed in Scilit:
- Adverse hepatic drug reactions: inflammatory episodes as consequence and contributorChemico-Biological Interactions, 2004
- Other biologic toxin bioweapons: ricin, staphylococcal enterotoxin B, and trichothecene mycotoxinsDermatologic Clinics, 2004
- Transcriptional and posttranscriptional roles for p38 mitogen-activated protein kinase in upregulation of TNF-α expression by deoxynivalenol (vomitoxin)Toxicology and Applied Pharmacology, 2003
- An Improved Method for the Purification of the Trichothecene Deoxynivalenol (Vomitoxin) from Fusarium graminearum CultureJournal of Agricultural and Food Chemistry, 2002
- The role of transcription factors in allergic inflammationJournal of Allergy and Clinical Immunology, 2002
- MAP Kinases in the Immune ResponseAnnual Review of Immunology, 2002
- MycotoxinsPublished by American Medical Association (AMA) ,2002
- Concurrent inflammation as a determinant of susceptibility to toxicity from xenobiotic agentsToxicology, 2001
- MAP kinase pathwaysProgress in Biophysics and Molecular Biology, 1999
- Endotoxemia in patients with alcoholic and non-alcoholic cirrhosis and in subjects with no evidence of chronic liver disease following acute alcohol excessJournal of Hepatology, 1987