Resistance of rat embryonic heart cells to the cytotoxic effects of cyclophosphamide does not involve aldehyde dehydrogenase‐mediated metabolism
- 1 April 1991
- journal article
- developmental pharmacology-and-teratology
- Published by Wiley in Teratology
- Vol. 43 (4), 307-318
- https://doi.org/10.1002/tera.1420430405
Abstract
No abstract availableThis publication has 20 references indexed in Scilit:
- Relationship of DNA damage and embryotoxicity induced by 4‐hydroperoxydechlorocyclophosphamide in postimplantation rat embryosTeratology, 1990
- Cell cycle analysis in the cardiac and neuroepithelial tissues of day 10 rat embryos and the effects of phosphoramide mustard, the major teratogenic metabolite of cyclophosphamideTeratology, 1989
- Uptake and binding of tritium from [chloroethyl3H] cyclophosphamide by rat embryos in vitroTeratology, 1985
- Induction and repair of macromolecular damage by alkylating agentsCancer Treatment Reviews, 1984
- Role of acrolein in cyclophosphamide teratogenicity in rat embryos in vitroToxicology and Applied Pharmacology, 1984
- The in vitro teratogenicity of cyclophosphamide in rat embryosTeratology, 1982
- Teratogenicity of cyclophosphamide metabolites: Phosphoramide mustard, acrolein, and 4-ketocyclophosphamide in rat embryos cultured in vitroToxicology and Applied Pharmacology, 1981
- Teratogenic bioactivation of cyclophosphamideLife Sciences, 1979
- A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye bindingAnalytical Biochemistry, 1976
- CELL DEATHS IN NORMAL VERTEBRATE ONTOGENYBiological Reviews, 1951