Flavivirus induces interferon-beta gene expression through a pathway involving RIG-I-dependent IRF-3 and PI3K-dependent NF-κB activation
- 1 January 2006
- journal article
- Published by Elsevier in Microbes and Infection
- Vol. 8 (1), 157-171
- https://doi.org/10.1016/j.micinf.2005.06.014
Abstract
No abstract availableKeywords
This publication has 70 references indexed in Scilit:
- Regulating Intracellular Antiviral Defense and Permissiveness to Hepatitis C Virus RNA Replication through a Cellular RNA Helicase, RIG-IJournal of Virology, 2005
- Novel roles of TLR3 tyrosine phosphorylation and PI3 kinase in double-stranded RNA signalingNature Structural & Molecular Biology, 2004
- Toll-like receptor signallingNature Reviews Immunology, 2004
- The RNA helicase RIG-I has an essential function in double-stranded RNA-induced innate antiviral responsesNature Immunology, 2004
- Hepatitis B viral HBx induces matrix metalloproteinase‐9 gene expression through activation of ERKs and PI‐3K/AKT pathways: Involvement of invasive potentialThe FASEB Journal, 2004
- Activation of the Interferon-βPromoter During Hepatitis C Virus RNA ReplicationViral Immunology, 2002
- Interferon regulatory factor-3 is an in vivo target of DNA-PKProceedings of the National Academy of Sciences, 2002
- Review: On the Role of IRF in Host DefenseJournal of Interferon & Cytokine Research, 2002
- The virus battles: IFN induction of the antiviral state and mechanisms of viral evasionCytokine & Growth Factor Reviews, 2001
- HOW CELLS RESPOND TO INTERFERONSAnnual Review of Biochemistry, 1998