The TOR nutrient signalling pathway phosphorylates NPR1 and inhibits turnover of the tryptophan permease
- 1 December 1998
- journal article
- Published by Springer Nature in The EMBO Journal
- Vol. 17 (23), 6924-6931
- https://doi.org/10.1093/emboj/17.23.6924
Abstract
EMBO Press is an editorially independent publishing platform for the development of EMBO scientific publications.Keywords
This publication has 48 references indexed in Scilit:
- Tor, a Phosphatidylinositol Kinase Homologue, Controls Autophagy in YeastJournal of Biological Chemistry, 1998
- cAMP Counter-regulates Insulin-mediated Protein Phosphatase-2A Inactivation in Rat Skeletal Muscle CellsPublished by Elsevier ,1996
- PIK-Related Kinases: DNA Repair, Recombination, and Cell Cycle CheckpointsScience, 1995
- NPI1, an essential yeast gene involved in induced degradation of Gap1 and Fur4 permeases, encodes the Rsp5 ubiquitin—protein ligaseMolecular Microbiology, 1995
- Target of rapamycin in yeast, TOR2, is an essential phosphatidylinositol kinase homolog required for G1 progressionCell, 1993
- Targets for Cell Cycle Arrest by the Immunosuppressant Rapamycin in YeastScience, 1991
- New yeast-Escherichia coli shuttle vectors constructed with in vitro mutagenized yeast genes lacking six-base pair restriction sitesGene, 1988
- Isolation of the NPRI gene responsible for the reactivatiion of ammonia‐sensitive amino‐acid permeases in Saccharomyces cerevisiaeEuropean Journal of Biochemistry, 1987
- A ten-minute DNA preparation from yeast efficiently releases autonomous plasmids for transformaion of Escherichia coliGene, 1987
- Study of the Positive Control of the General Amino‐Acid Permease and Other Ammonia‐Sensitive Uptake Systems by the Product of the NPR1 Gene in the Yeast Saccharomyces cerevisiaeEuropean Journal of Biochemistry, 1983