Increased α1(I) Procollagen Gene Expression in Tight Skin (TSK) Mice Myocardial Fibroblasts Is Due to a Reduced Interaction of a Negative Regulatory Sequence with AP-1 Transcription Factor
Open Access
- 1 April 1995
- journal article
- Published by Elsevier
- Vol. 270 (16), 9313-9321
- https://doi.org/10.1074/jbc.270.16.9313
Abstract
No abstract availableKeywords
This publication has 26 references indexed in Scilit:
- Phenotype suppression: A postulated molecular mechanism for mediating the relationship of proliferation and differentiation by Fos/Jun interactions at AP‐1 sites in steroid responsive promoter elements of tissue‐specific genesJournal of Cellular Biochemistry, 1991
- Transcriptional and posttranscriptional regulation of tumor necrosis factor gene expression in human monocytes.JCI Insight, 1988
- Purified transcription factor AP-1 interacts with TPA-inducible enhancer elementsCell, 1987
- Phorbol ester-inducible genes contain a common cis element recognized by a TPA-modulated trans-acting factorCell, 1987
- Nucleotide sequence of a cDNA clone for mouse proα 1(I) collagen proteinGene, 1984
- Scleroderma‐like alterations in collagen metabolism occurring in the TSK (tight skin) mouseArthritis & Rheumatism, 1984
- Biochemical Characterization of Collagens and of a Non-Collagenous Protein Synthesized by Guinea Pig Lung Fibroblasts in CultureConnective Tissue Research, 1983
- A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye bindingAnalytical Biochemistry, 1976
- Collagen biosynthesis during connective tissue development in chick embryoDevelopmental Biology, 1972
- Use of a mixture of proteinase-free collagenases for the specific assay of radioactive collagen in the presence of other proteinsBiochemistry, 1971