Evidence for a Functional Role of the Molecular Chaperone Clusterin in Amyloidotic Cardiomyopathy
- 1 January 2011
- journal article
- research article
- Published by Elsevier in The American Journal of Pathology
- Vol. 178 (1), 61-68
- https://doi.org/10.1016/j.ajpath.2010.11.015
Abstract
No abstract availableKeywords
Funding Information
- National Institutes of Health (R01AG031804)
- Walk for An Angel and Jamieson Funds for Amyloid Research
- NIH (HL079099, HL095891, HL102631)
This publication has 22 references indexed in Scilit:
- Amyloidogenic light chains induce cardiomyocyte contractile dysfunction and apoptosis via a non-canonical p38α MAPK pathwayProceedings of the National Academy of Sciences, 2010
- Amyloid heart disease. New frontiers and insights in pathophysiology, diagnosis, and management.2005
- Human Amyloidogenic Light Chains Directly Impair Cardiomyocyte Function Through an Increase in Cellular Oxidant StressCirculation Research, 2004
- Clusterin is a secreted mammalian chaperoneTrends in Biochemical Sciences, 2000
- Clusterin Has Chaperone-like Activity Similar to That of Small Heat Shock ProteinsJournal of Biological Chemistry, 1999
- Stress-induced transcription of the clusterin/apoJ geneBiochemical Journal, 1997
- Apolipoprotein J/clusterin induction in myocarditis: A localized response gene to myocardial injury.1996
- Clusterin: Physiologic and pathophysiologic considerationsThe International Journal of Biochemistry & Cell Biology, 1995
- Apolipoprotein J expression at fluid-tissue interfaces: potential role in barrier cytoprotection.Proceedings of the National Academy of Sciences, 1993
- Echocardiographic assessment of left ventricular hypertrophy: Comparison to necropsy findingsThe American Journal of Cardiology, 1986