Chk2 is required for HSV-1 ICP0-mediated G2/M arrest and enhancement of virus growth
- 5 March 2008
- Vol. 375 (1), 13-23
- https://doi.org/10.1016/j.virol.2008.01.038
Abstract
No abstract availableThis publication has 68 references indexed in Scilit:
- Using or abusing: viruses and the cellular DNA damage responseTrends in Microbiology, 2007
- ATR dependent activation of Chk2Journal of Cellular Physiology, 2006
- Reciprocal Activities between Herpes Simplex Virus Type 1 Regulatory Protein ICP0, a Ubiquitin E3 Ligase, and Ubiquitin-Specific Protease USP7Journal of Virology, 2005
- Inhibition of the Herpes Simplex Virus Type 1 DNA Polymerase Induces Hyperphosphorylation of Replication Protein A and Its Accumulation at S-Phase-Specific Sites of DNA Damage during InfectionJournal of Virology, 2005
- Questioning the Role of Checkpoint Kinase 2 in the p53 DNA Damage ResponsePublished by Elsevier ,2003
- The Chk2 Tumor Suppressor Is Not Required for p53 Responses in Human Cancer CellsPublished by Elsevier ,2003
- DNA damage activates ATM through intermolecular autophosphorylation and dimer dissociationNature, 2003
- Degradation of Nucleosome-associated Centromeric Histone H3-like Protein CENP-A Induced by Herpes Simplex Virus Type 1 Protein ICP0Journal of Biological Chemistry, 2001
- A human homologue of the checkpoint kinase Cds1 directly inhibits Cdc25 phosphataseCurrent Biology, 1999
- Enhancement of host cell reactivation of ultraviolet-irradiated herpes simplex virus by caffeine, hydroxyurea and 5-bromodeoxyuridineInternational Journal of Cancer, 1979